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Scientific collaboration between Professor Marco Toscani and Dr. Pasquale Fino, Chair of Plastic, Reconstructive and Aesthetic Surgery, Umberto I Health Center – “Sapienza” University of Rome.

Scalp pain and irritation (or trichodynia) are pathological conditions that affect both women (in a greater percentage) and men (in a lower percentage). The pain felt on the scalp, spontaneous or caused by treatment, and in particular around the roots of the hair, may be more or less intense, alternating at times between periods of disappearance, associated with burning, tingling or itching.

Usually, this particular scalp condition does is not located in a specific area and increases when hair is touched or brushed. Often, trichodynia is associated with hair loss. A correlation can be found between telogen effluvium and trichodynia and between seborrheic hair loss and the constant sensation of scalp pain or irritation.
Trichodynia is classified among the allodynia, conditions in which pain is felt on the skin without recognizable external causes or appears to be caused by usually innocuous stimuli. Patients suffering from this condition perceive a burning sensation and irritation on the scalp. The itchiness is so intense that thay are forced to scratch their head. The relief however is very short-lasting.
The reduction of the activation threshold of nerve endings (nociceptors), whose endings spread over the entire scalp (the pilosebaceous apparatus of each hair and the correspondent nerve endings at the level of the skin), seems to have a decisive role in triggering allodynia. The inflammation of the skin activates the nociceptors that secrete neuropeptide P, the neurotransmitter of pain. Individuals who notice this condition tend to scratch their head, pull out their hair hair to feel mild relief, but this leads to hair loss due to trichotillomania.
Although the (aforementioned) chemical process that causes skin inflammation is now known, the causes are still unclear, namely what triggers this process and how the nociceptors act. There is a strong correlation between the emotional component and trichodynia and between trichodynia and hair loss. Nervousness, stress and anxiety are associated with the appearance of scalp pain and itchiness, and with subsequent hair loss.

Another cause that triggers pain is hyperseborrhea, which leads to seborrheic dermatitis and the appearance of pain, inflammation and skin irritation. Another cause is greasy dandruff, which irritates the scalp, damaging follicular activity. Other possible causes of pain are scalp folliculitis and acne that lead to rashes, pain and itchy skin.
During the inflammatory process, the fact that the muscles of the arrector pili of the hair are forced into a strange position for long period of time may cause pain. The arrector pili muscles and the hair follicles may then become inflamed, thereby triggering the inflammatory process of the skin. Another cause of trichodynia may be neuro-muscular inflammation of skin overstressed by the tendency to grind one’s teeth, especially at night (bruxism), with the subsequent contraction of the temporomandibular joint that, with the passing of time, may involve the cranial muscles, causing headaches and trichodynia. Trichodynia should be cared for and countered with trichological treatments that aim to eliminate the underlying causes, such as hyperseborrhea and dandruff. The use of lotions and shampoos that cleanse the skin of impurities and bacteria that cause inflammation and conseuquent pain is recommended. It is also important to use remedies against hair loss.

Suitable specific and personalized therapies should be used only after a careful examination of the hair and a scrupulous skin analysis. The following treatments have proven to be useful:

1) the use of laser treatments that increase the blood capillary vasodilation necessary for providing sustenance to hair bulbs

2) treatments with high frequency rays that clean, cleanse and disinfect the scalp, improving blood supply and stimulating healthy hair growth

3) hair massages that allow follicles to expel a small amount of sebum that accumulates and facilitates the relaxation of the skin and scalp muscles.

Scalp psoriasis

La psoriasi è una frequente dermatosi cronica eritemo-squamosa con squama tipica (psoriasica), circoscritta o diffusa.
La psoriasi è un problema del cuoio capelluto non annoverata tra le cause che comportano la caduta dei capelli.

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Sebaceous glands

The sebaceous glands are glands that secrete sebum, an oily, acidic substance with a pH of 3.5. They are connected laterally to the hair follicle. Sebaceous glands are found with a density of approximately 100/cm2 throughout all areas of the skin. In the human body, they are located across the entire surface of the skin, except for the palms of the hands and the soles of the feet.

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Hyperseborrhea

Hyperseborrhea or hypersecretion of sebum is simply a scalp problem due to an excessive production of sebum caused by hyperactivity of the sebaceous glands. Immediate symptoms of hyperseborrhea are scalp itchiness and pain. However, a later symptom is hair loss.

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Scalp pain

Scalp pain and irritation (or trichodynia) are pathological conditions that affect both women (in a greater percentage) and men (in a lower percentage). The pain felt on the scalp, spontaneous or caused by treatment, and in particular around the roots of the hair, may be more or less intense, alternating at times between periods of disappearance, associated with burning, tingling or itching.

Read more »

Hair loss (telogen defluvium)

With the term Telogen Defluvium we mean a modest, not excessive, loss of hair in the telogen phase, but which tends to often be irreversible, with the precise characteristics of hair in decay or involution. The hair that falls out is usually short and fine, with bulbs that are undeveloped and reduced in size.

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Hair follicles and bulbs

A hair follicle is a formation of epidermal origin, anchored in the dermis and consisting of a hair and its sheath.
The formation of hair follicles occurs early. They begin to appear as early as the second month of gestation. These follicles will continue to form for the entire duration of the pregnancy. At birth, the baby will indeed have the entire supply of follicles with which it was genetically equipped and from this moment on, no others will form.

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