Scientific collaboration between Professor Marco Toscani and Dr. Pasquale Fino, Chair of Plastic, Reconstructive and Aesthetic Surgery, Umberto I Health Center – “Sapienza” University of Rome.
Seborrheic dermatitis is a very common scalp condition characterized by the presence of yellow and oily flakes on the skin.
It is associated with erythema, small scaly, and intense itching.
Seborrheic dermatitis is a well-known condition but is still not easily identifiable. Many men and women starting from puberty have oily, greasy, shiny and thickened skin with large hair follicles, especially noticeable around the nose and mouth, forehead, torso, scalp (affected by so-called oily dandruff).
Seborrheic dermatitis appears in these individuals. It is characterized by red, reddish-yellow or dark red skin patches, covered with small and medium sized, thick and greasy flakes, sometimes appearing as scaly formations with blisters that are not easily noticed.
Seborrheic eczema is a typical form surrounded by seborrheic dermatitis limited to certain areas of the body, found at the level of the sternum and shoulder blades, characterized by various round patches with clearly defined convex edges. Another form is characterized by pityriasiform and psoriasiform patches. Complications of seborrheic dermatitis are: eczematization (the most common complication), external otitis, occipital and nuchal eczema, umbilical eczema, areola eczema in women and lastly perineum-genital eczema.
Often, seborrheic dermatitis is located on the scalp and along its edges. An irregular patch often emerges from the scalp onto the forehead called a “seborrheic crown”. It is important to remember that seborrheic dermatitis in and of itself does not cause hair loss.
The etiopathogenesis of seborrheic dermatitis is not completely clear. Recognized concurrent causes are bacterial, fungal (Malassezia), infectious, mechanical, irritative, psychosomatic and sebaceous dysfunction (most probably of genetic origin) factors. In the case of seborrheic dermatitis sebum undergoes a chemical transformation. In fact, a reduction of triglycerides, squalene and cholesterol occurs.
Hair treatments like lotions and shampoos are commonly used as local anti-seborrheic treatments. Non-halogenated corticosteroids in the form of gels or lotions are very effective. Antibiotics and antifungal treatments may also be used. Positive results have also been achieved by using retinoic acid and ketoconazole, general purpose antifungals.
Hyperseborrhea or hypersecretion of sebum is simply a scalp problem due to an excessive production of sebum caused by hyperactivity of the sebaceous glands. Immediate symptoms of hyperseborrhea are scalp itchiness and pain. However, a later symptom is hair loss.
Changes in the hair shaft lead to deterioration with the appearance of ruined or damaged hair.
In healthy hair, the cuticle is whole, with overlapping shingles. Hair with whole ends appears shiny, elastic and brushes easily.
Dry skin or asteatosis is a scalp condition deriving from a lipid deficiency.
It is often associated with a situation of exacerbated dehydration. We can identify both internal and external causes for this anomaly.
Genetic factors influence and determine the size of the diameter in hair.
Its volume varies from person to person. This is why some individuals are born with thin hair, while others are born with thick hair.
La psoriasi è una frequente dermatosi cronica eritemo-squamosa con squama tipica (psoriasica), circoscritta o diffusa.
La psoriasi è un problema del cuoio capelluto non annoverata tra le cause che comportano la caduta dei capelli.
A hair follicle is a formation of epidermal origin, anchored in the dermis and consisting of a hair and its sheath.
The formation of hair follicles occurs early. They begin to appear as early as the second month of gestation. These follicles will continue to form for the entire duration of the pregnancy. At birth, the baby will indeed have the entire supply of follicles with which it was genetically equipped and from this moment on, no others will form.
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